Brain with Alzheimer’s disease. This image shows a comparison between a healthy brain and one affected by Alzheimer’s, highlighting brain tissue loss. In this representation, the cerebral hemispheres, neurons, beta-amyloid plaques, tau neurofibrillary tangles, and cerebral cortex are identified. These structures enable memory, thinking, and cognitive functions.
The brain with Alzheimer’s disease has an altered function in the nervous system. Moreover, under normal conditions, neurons maintain efficient communication. Additionally, they support learning and memory. Furthermore, they sustain normal cognitive functions.
On the other hand, in Alzheimer’s disease, beta-amyloid plaques accumulate between neurons. These plaques interfere with neuronal communication. Moreover, tau protein forms neurofibrillary tangles within cells. Additionally, these changes damage neuronal structure.
Transport of nerve signals occurs through synapses. For example, electrical impulses allow information transmission. Moreover, when neurons deteriorate, this process is affected. Additionally, communication becomes inefficient.
Key processes include neuronal degeneration and abnormal protein accumulation. Moreover, memory loss appears as a primary symptom. However, cognitive impairments are also present. Additionally, abilities such as language and orientation are affected.
Regulation depends on genetic and environmental factors. Moreover, aging is the main risk factor. Additionally, other factors may accelerate brain deterioration. Furthermore, progression is gradual.
As a result, brain volume progressively decreases. Additionally, patient independence declines. Moreover, daily activities become compromised.
Therefore, the brain with Alzheimer’s disease reflects the structural impact of this condition. In conclusion, protein accumulation causes neuronal damage and progressive cognitive decline.